A recurrent mutation in KCNA2 as a novel cause of hereditary spastic paraplegia and ataxia

نویسندگان

  • Katherine L. Helbig
  • Ulrike B.S. Hedrich
  • Deepali N. Shinde
  • Ilona Krey
  • Anne‐Christin Teichmann
  • Julia Hentschel
  • Julian Schubert
  • Adam C. Chamberlin
  • Robert Huether
  • Hsiao‐Mei Lu
  • Wendy A. Alcaraz
  • Sha Tang
  • Chelsy Jungbluth
  • Sarah L. Dugan
  • Leena Vainionpää
  • Kathrin N. Karle
  • Matthis Synofzik
  • Ludger Schöls
  • Rebecca Schüle
  • Anna‐Elina Lehesjoki
  • Ingo Helbig
  • Holger Lerche
  • Johannes R. Lemke
چکیده

The hereditary spastic paraplegias (HSPs) are heterogeneous neurodegenerative disorders with over 50 known causative genes. We identified a recurrent mutation in KCNA2 (c.881G>A, p.R294H), encoding the voltage-gated K(+) -channel, KV 1.2, in two unrelated families with HSP, intellectual disability (ID), and ataxia. Follow-up analysis of > 2,000 patients with various neurological phenotypes identified a de novo p.R294H mutation in a proband with ataxia and ID. Two-electrode voltage-clamp recordings of Xenopus laevis oocytes expressing mutant KV 1.2 channels showed loss of function with a dominant-negative effect. Our findings highlight the phenotypic spectrum of a recurrent KCNA2 mutation, implicating ion channel dysfunction as a novel HSP disease mechanism. Ann Neurol 2016.

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عنوان ژورنال:

دوره 80  شماره 

صفحات  -

تاریخ انتشار 2016